Product References
Investigation into the role of the MITA-TRIM38 interaction in regulating pyroptosis and maintaining immune tolerance at the maternal-fetal interface.
Cell death & disease
Liu J,Deng Y,Wang A,Liu B,Zhou X,Yin T,Wang Y,Tang T,Qiu Y,Chen J,Yang J
MA5-26235 was used in Immunohistochemistry to indicate that pyroptosis played an important role in hindering the transformation of M1 to M2 and maintaining the immunosuppressed state of the maternal-fetal interface.
Tue Nov 28 00:00:00 UTC 2023
Investigation into the role of the MITA-TRIM38 interaction in regulating pyroptosis and maintaining immune tolerance at the maternal-fetal interface.
Cell death & disease
Liu J,Deng Y,Wang A,Liu B,Zhou X,Yin T,Wang Y,Tang T,Qiu Y,Chen J,Yang J
MA5-26235 was used in Immunohistochemistry to indicate that pyroptosis played an important role in hindering the transformation of M1 to M2 and maintaining the immunosuppressed state of the maternal-fetal interface.
Tue Nov 28 00:00:00 UTC 2023
Investigation into the role of the MITA-TRIM38 interaction in regulating pyroptosis and maintaining immune tolerance at the maternal-fetal interface.
Cell death & disease
Liu J,Deng Y,Wang A,Liu B,Zhou X,Yin T,Wang Y,Tang T,Qiu Y,Chen J,Yang J
MA5-26235 was used in Immunohistochemistry to indicate that pyroptosis played an important role in hindering the transformation of M1 to M2 and maintaining the immunosuppressed state of the maternal-fetal interface.
Tue Nov 28 00:00:00 UTC 2023
Investigation into the role of the MITA-TRIM38 interaction in regulating pyroptosis and maintaining immune tolerance at the maternal-fetal interface.
Cell death & disease
Liu J,Deng Y,Wang A,Liu B,Zhou X,Yin T,Wang Y,Tang T,Qiu Y,Chen J,Yang J
MA5-26235 was used in Immunohistochemistry to indicate that pyroptosis played an important role in hindering the transformation of M1 to M2 and maintaining the immunosuppressed state of the maternal-fetal interface.
Tue Nov 28 00:00:00 UTC 2023
TRIM38 protects H9c2 cells from hypoxia/reoxygenation injury via the TRAF6/TAK1/NF-κB signalling pathway.
PeerJ
Lu Z,Deng M,Ma G,Chen L
MA5-26235 was used in Western Blotting to establish an in vitro cellular hypoxia/reperfusion (H/R) model to explore the role and potential mechanisms of TRIM38 in H9c2, a rat cardiomyoblast cell line.
Tue Jan 17 00:00:00 UTC 2023
TRIM38 protects H9c2 cells from hypoxia/reoxygenation injury via the TRAF6/TAK1/NF-κB signalling pathway.
PeerJ
Lu Z,Deng M,Ma G,Chen L
MA5-26235 was used in Western Blotting to establish an in vitro cellular hypoxia/reperfusion (H/R) model to explore the role and potential mechanisms of TRIM38 in H9c2, a rat cardiomyoblast cell line.
Tue Jan 17 00:00:00 UTC 2023
Tripartite motif 38 attenuates cardiac fibrosis after myocardial infarction by suppressing TAK1 activation via TAB2/3 degradation.
iScience
Lu Z,Hao C,Qian H,Zhao Y,Bo X,Yao Y,Ma G,Chen L
MA5-26235 was used in Western Blotting to help develop therapeutic strategies to treat and prevent cardiac fibrosis.
Fri Aug 19 00:00:00 UTC 2022
Tripartite motif 38 attenuates cardiac fibrosis after myocardial infarction by suppressing TAK1 activation via TAB2/3 degradation.
iScience
Lu Z,Hao C,Qian H,Zhao Y,Bo X,Yao Y,Ma G,Chen L
MA5-26235 was used in Western Blotting to help develop therapeutic strategies to treat and prevent cardiac fibrosis.
Fri Aug 19 00:00:00 UTC 2022